Hi, I am Pritha Bhattacharjee, My LiveDNA is 91.12118
 
   
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Dr. Pritha Bhattacharjee
 
Highest Degree: PostDoc Fellow in Molecular Genetics from Indian Institute of Chemical Biology, India
 
Institute: University of Calcutta, India
 
Area of Interest: Molecular Sciences
  •   Toxicology
  •   Molecular Genetics
  •   Environmental Health
  •   Zoology
 
URL: http://livedna.org/91.12118
 
My SELECTED Publications
1:   Banerjee, M., N. Banerjee, P. Bhattacharjee, D. Mondal and P.R. Lythgoe et al., 2013. High arsenic in rice is associated with elevated genotoxic effects in humans. Sci. Rep., Vol. 3. 10.1038/srep02195.
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2:   Banerjee, M., N. Banerjee, P. Ghosh, J.K. Das and S. Basu et al., 2010. Evaluation of the serum catalase and myeloperoxidase activities in chronic arsenic-exposed individuals and concomitant cytogenetic damage. Toxicol. Appl. Pharmacol., 249: 47-54.
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3:   Basu, A., P. Ghosh, J.K. Das, A. Banerjee, K. Ray and A.K. Giri, 2004. Micronuclei as biomarkers of carcinogen exposure in populations exposed to arsenic through drinking water in west Bengal, India: A comparative study in three cell types. Cancer Epidemiol. Biomarkers Prev., 13: 820-827.
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4:   Bhattacharjee, P. and S. Paul, 2016. Risk of occupational exposure to asbestos, silicon and arsenic on pulmonary disorders: Understanding the genetic-epigenetic interplay and future prospects. Environ. Res., 147: 425-434.
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5:   Bhattacharjee, P., D. Chatterjee, K.K. Singh and A.K. Giri, 2013. Systems biology approaches to evaluate arsenic toxicity and carcinogenicity: An overview. Int. J. Hyg. Environ. Health, 216: 574-586.
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6:   Bhattacharjee, P., M. Banerjee and A.K. Giri, 2013. Role of genomic instability in arsenic-induced carcinogenicity: A review. Environ. Int., 53: 29-40.
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7:   Bhattacharjee, P., N. Das, D. Chatterjee, A. Banerjee and J.K. Das et al., 2013. Association of NALP2 polymorphism with arsenic induced skin lesions and other health effects. Mutat. Res. Genet. Toxicol. Environ. Mutagen., 755: 1-5.
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8:   Bhattacharjee, P., S. Paul, M. Banerjee, D. Patra and P. Banerjee et al., 2013. Functional compensation of glutathione S-transferase M1 (GSTM1) null by another GST superfamily member, GSTM2. Sci. Rep., 10.1038/srep02704.
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9:   Biswas, A., T. Sadhukhan, K. Bose, P. Ghosh and A.K. Giri et al., 2012. Role of glutathione S-transferase T1, M1 and P1 polymorphisms in Indian Parkinson's disease patients. Parkinsonism Related Disorders, 18: 664-665.
10:   Biswas, R., P. Ghosh, N. Banerjee, J.K. Das and T. Sau et al., 2008. Analysis of T-cell proliferation and cytokine secretion in the individuals exposed to arsenic. Hum. Exp. Toxicol., 27: 381-386.
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11:   Chatterjee, D., P. Bhattacharjee, T.J. Sau, J.K. Das and N. Sarma et al., 2015. Arsenic exposure through drinking water leads to senescence and alteration of telomere length in humans: A case-control study in west Bengal, India. Mol. Carcinog., 54: 800-809.
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12:   De Chaudhuri, S., P. Ghosh, N. Sarma, P. Majumdar and T.J. Sau et al., 2008. Genetic variants associated with arsenic susceptibility: study of purine nucleoside phosphorylase, arsenic (+ 3) methyltransferase, and glutathione S-transferase omega genes. Environ. Health Perspect., 116: 501-505.
PubMed  |  
13:   Ghosh, P., A. Basu, K.K. Singh and A.K. Giri, 2008. Evaluation of cell types for assessment of cytogenetic damage in arsenic exposed population. Mol. Cancer, Vol. 7. 10.1186/1476-4598-7-45.
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14:   Ghosh, P., M. Banerjee, A.K. Giri and K. Ray, 2008. Toxicogenomics of arsenic: Classical ideas and recent advances. Mutat. Res. Rev. Mutat. Res., 659: 293-301.
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15:   Ghosh, P., M. Banerjee, S. De Chaudhuri, J.K. Das, N. Sarma, A. Basu and A.K. Giri, 2007. Increased chromosome aberration frequencies in the Bowen's patients compared to non-cancerous skin lesions individuals exposed to arsenic. Mutat. Res. Genet. Toxicol. Environ. Mutagen., 632: 104-110.
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16:   Ghosh, P., M. Banerjee, S. De Chaudhuri, R. Chowdhury and J.K. Das et al., 2007. Comparison of health effects between individuals with and without skin lesions in the population exposed to arsenic through drinking water in west Bengal, India. J. Exposure Sci. Environ. Epidemiol., 17: 215-223.
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17:   Gosh, P., A. Basu, J. Mahata, S. Basu and M. Sengupta et al., 2006. Cytogenetic damage and genetic variants in the individuals susceptible to arsenic-induced cancer through drinking water. Int. J. Cancer, 118: 2470--2478.
PubMed  |  
18:   Kundu, M., P. Ghosh, S. Mitra, J.K. Das and T.J. Sau et al., 2011. Precancerous and non-cancer disease endpoints of chronic arsenic exposure: The level of chromosomal damage and XRCC3 T241M polymorphism. Mutat. Res. Fundam. Mol. Mech. Mutagen., 706: 7-12.
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19:   Mahata, J., M. Chaki, P. Ghosh, L.K. Das and K. Baidya et al., 2004. Chromosomal aberrations in arsenic-exposed human populations: A review with special reference to a comprehensive study in west Bengal, India. Cytogenet. Genome Res., 104: 359-364.
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20:   Mahata, J., P. Ghosh, J.N. Sarkar, K. Ray, A.T. Natarajan and A.K. Giri, 2004. Effect of sodium arsenite on peripheral lymphocytes in vitro: Individual susceptibility among a population exposed to arsenic through the drinking water. Mutagen., 19: 223-229.
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21:   Paul, S., N. Das, P. Bhattacharjee, M. Banerjee and J.K. Das et al., 2013. Arsenic-induced toxicity and carcinogenicity: A two-wave cross-sectional study in arsenicosis individuals in west Bengal, India. J. Exposure Sci. Environ. Epidemiol., 23: 156-162.
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22:   Paul, S., P. Bhattacharjee, P.K. Mishra, D. Chatterjee and A. Biswas et al., 2013. Human urothelial micronucleus assay to assess genotoxic recovery by reduction of arsenic in drinking water: A cohort study in west Bengal, India. Biometals, 26: 855-862.
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