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2. Chougule, R.A., J.U. Kazi and L. Ronnstrand, 2016. FYN expression potentiates FLT3-ITD induced STAT5 signaling in acute myeloid leukemia. Oncotarget. 10.18632/oncotarget.7128.
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3. Chougule, R.A., E. Cordero, S.A. Moharram, K. Pietras, L. Ronnstrand and J.U. Kazi, 2016. Expression of GADS enhances FLT3-induced mitogenic signaling. Oncotarget. 10.18632/oncotarget.7415.
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4. Alam, M.W., C.U. Persson, S. Reinbothe, J.U. Kazi and L. Ronnstrand et al., 2016. HIF2α contributes to antiestrogen resistance via positive bilateral crosstalk with EGFR in breast cancer cells. Oncotarget. 10.18632/oncotarget.7167.
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5. Zhang, J., O. Vakhrusheva, S.R. Bandi, O. Demirel and J.U. Kazi et al., 2015. The phosphatases STS1 and STS2 regulate hematopoietic stem and progenitor cell fitness. Stem Cell Rep., 5: 633-646.
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6. Lindblad, O., T. Li, X. Su, J. Sun and N.N. Kabir et al., 2015. BEX1 acts as a tumor suppressor in acute myeloid leukemia. Oncotarget, 6: 21395-21405.
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7. Lindblad, O., R.A. Chougule, S.A. Moharram, N.N. Kabir, J. Sun, J.U. Kazi and L. Ronnstrand, 2015. The role of HOXB2 and HOXB3 in acute myeloid leukemia. Biochem. Biophys. Res. Commun., 467: 742-747.
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8. Lindblad, O., J.U. Kazi, L. Ronnstrand and J. Sun, 2015. PI3 kinase is indispensable for oncogenic transformation by the V560D mutant of c-Kit in a kinase-independent manner. Cell. Mol. Life Sci., 72: 4399-4407.
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9. Kazi, J.U., N.N. Kabir and L. Ronnstrand, 2015. Role of SRC-Like Adaptor Protein (SLAP) in immune and malignant cell signaling. Cell. Mol. Life Sci., 72: 2535-2544.
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10. Kazi, J.U., N.N. Kabir and L. Ronnstrand, 2015. Brain-Expressed X-linked (BEX) proteins in human cancers. Biochim. Biophys. Acta (BBA)-Rev. Cancer, 1856: 226-233.
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11. Kazi, J.U., A. Flores-Morales and L. Ronnstrand, 2015. SOCS6 (suppressor of cytokine signaling 6). Atlas Genet. Cytogenet. Oncol. Haematol., 19: 50-52.
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12. Agarwal, S., J.U. Kazi, S. Mohlin, S. Pahlman and L. Ronnstrand, 2015. The activation loop tyrosine 823 is essential for the transforming capacity of the c-Kit oncogenic mutant D816V. Oncogene, 34: 4581-4590.
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13. Sun, J., S. Mohlin, A. Lundby, J.U. Kazi and U. Hellman et al., 2014. The PI3-kinase isoform p110δ is essential for cell transformation induced by the D816V mutant of c-Kit in a lipid-kinase-independent manner. Oncogene, 33: 5360-5369.
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14. Puissant, A., N. Fenouille, G. Alexe, Y. Pikman and C.F. Bassil et al., 2014. SYK Is a critical regulator of FLT3 in acute myeloid leukemia. Cancer Cell, 25: 226-242.
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15. Kazi, J.U., S. Agarwal, J. Sun, E. Bracco and L. Ronnstrand, 2014. Src-Like Adaptor Protein (SLAP) differentially regulates normal and oncogenic c-Kit signaling. J. Cell Sci., 127: 653-662.
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16. Kazi, J.U., N.N. Kabir, A. Flores-Morales and L. Ronnstrand, 2014. SOCS proteins in regulation of receptor tyrosine kinase signaling. Cell. Mol. Life Sci., 71: 3297-3310.
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17. Kabir, N.N., L. Ronnstrand and J.U. Kazi, 2014. Keratin 19 expression correlates with poor prognosis in breast cancer. Mol. Biol. Rep., 41: 7729-7735.
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18. Kabir, N.N., J. Sun, L. Ronnstrand and J.U. Kazi, 2014. SOCS6 is a selective suppressor of receptor tyrosine kinase signaling. Tumor Biol., 35: 10581-10589.
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19. Kabir, N.N. and J.U. Kazi, 2014. Grb10 is a dual regulator of receptor tyrosine kinase signaling. Mol. Biol. Rep., 41: 1985-1992.
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20. Kazi, J.U., N.N. Kabir and L. Ronnstrand, 2013. Protein Kinase C (PKC) as a drug target in chronic lymphocytic leukemia. Med. Oncol., 30: 1-8.
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21. Kazi, J.U., M. Vaapil, S. Agarwal, E. Bracco, S. Pahlman and L. Ronnstrand, 2013. The tyrosine kinase CSK associates with FLT3 and c-Kit receptors and regulates downstream signaling. Cell. Signal., 25: 1852-1860.
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22. Kazi, J.U. and L. Ronnstrand, 2013. Suppressor of cytokine signaling 2 (SOCS2) associates with FLT3 and negatively regulates downstream signaling. Mol. Oncol., 7: 693-703.
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23. Kazi, J.U. and L. Ronnstrand, 2013. FLT3 signals via the adapter protein Grb10 and overexpression of Grb10 leads to aberrant cell proliferation in acute myeloid leukemia. Mol. Oncol., 7: 402-418.
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24. Kabir, N.N., L. Ronnstrand and J.U. Kazi, 2013. The basic helix-loop-helix (bHLH) proteins in breast cancer progression. Med. Oncol., 30: 1-3.
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25. Kabir, N.N., L. Ronnstrand and J.U. Kazi, 2013. Protein Kinase C (PKC) expression is deregulated in chronic lymphocytic leukemia. Leukemia Lymphoma, 54: 2288-2290.
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26. Kabir, N.N., L. Ronnstrand and J.U. Kazi, 2013. FLT3 mutations in patients with childhood Acute Lymphoblastic Leukemia (ALL). Med. Oncol., 30: 462-462.
27. Kabir, N.N., L. Ronnstrand and J.U. Kazi, 2013. Deregulation of protein phosphatase expression in acute myeloid leukemia. Med. Oncol., 30: 1-6.
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28. Kabir, N.N. and J.U. Kazi, 2013. Selective mutation in ATP-binding site reduces affinity of drug to the kinase: A possible mechanism of chemo-resistance. Med. Oncol., 30: 448-448.
29. Agarwal, S., J.U. Kazi and L. Ronnstrand, 2013. Phosphorylation of the activation loop tyrosine 823 in c-Kit is crucial for cell survival and proliferation. J. Biol. Chem., 288: 22460-22468.
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30. Lin, D.C., T. Yin, M. Koren-Michowitz, L.W. Ding and S. Gueller et al., 2012. Adaptor protein Lnk binds to and inhibits normal and leukemic FLT3. J. Am. Soc. Hematol., 120: 3310-3317.
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31. Kazi, J.U., J. Sun, B. Phung, F. Zadjali and A. Flores-Morales et al., 2012. Suppressor of Cytokine Signaling 6 (SOCS6) Negatively Regulates Flt3 Signal Transduction Through Direct Binding to Phosphorylated Tyrosines 591 and 919 of Flt3. J. Biol. Chem., 287: 36509-36517.
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32. Kazi, J.U. and L. Ronnstrand, 2012. Src-Like Adaptor Protein (SLAP) binds to the receptor tyrosine kinase Flt3 and modulates receptor stability and downstream signaling. PLoS One, Vol. 7. 10.1371/journal.pone.0053509.
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33. Khan, M.M.G., A.M. Ryden, M.S. Chowdhury, M.A. Hasan and J.U. Kazi, 2011. Maximum likelihood analysis of mammalian p53 indicates the presence of positively selected sites and higher tumorigenic mutations in purifying sites. Gene, 483: 29-35.
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34. Kazi, J.U., 2011. The mechanism of protein kinase C regulation. Front Biol., 6: 328-336.
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35. Kabir, N.N., M.I. Hossain and J.U. Kazi, 2011. Comparative studies on human and rat basic Helix-loop-helix proteins. Asian J. Biol. Sci., 4: 601-608.
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36. Kabir, N.N. and J.U. Kazi, 2011. Comparative analysis of human and bovine protein kinases reveals unique relationship and functional diversity. Genet. Mol. Biol., 34: 587-591.
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37. Kim, H.J., J.U. Kazi, Y.R. Lee, D.H. Nguyen and H.B. Lee et al., 2010. Visualization of the melanosome transfer-inhibition in a mouse epidermal cell co-culture model. Int. J. Mol. Med., 25: 249-253.
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38. Kazi, J.U., C.R. Kim and J.W. Soh, 2009. Subcellular localization of diacylglycerol-responsive protein kinase C isoforms in HeLa cells. Bull. Korean Chem. Soc., 30: 1981-1984.
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39. Soh, J.W., J.U. Kazi, H. Li, W.J. Thompson and I.B. Weinstein, 2008. Celecoxib-induced growth inhibition in SW480 colon cancer cells is associated with activation of protein kinase G. Mol. Carcinogenesis, 47: 519-525.
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40. Kazi, J.U., N.N. Kabir and J.W. Soh, 2008. Bioinformatic prediction and analysis of eukaryotic protein kinases in the rat genome. Gene, 410: 147-153.
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41. Kazi, J.U. and J.W. Soh, 2008. Role of regulatory domain mutants of PKC isoforms in c-fos induction. Bull. Korean Chem. Soc., 29: 252-254.
42. Kazi, J.U. and J.W. Soh, 2008. Induction of the nuclear proto-oncogene c-fos by the phorbol ester TPA and c-H-Ras. Mol. Cells, 26: 462-467.
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43. Kazi, J.U. and J.W. Soh, 2007. Isoform-specific translocation of PKC isoforms in NIH3T3 cells by TPA. Biochem. Biophys. Res. Commun., 364: 231-237.
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